Migraine headaches are debilitating, primary headaches that cause a myriad of neurological symptoms. Although still not fully understood, migraine headaches are estimated to affect nearly 15% of the population.
Migraine headaches are true, primary headaches affecting the brain and the surrounding tissues. There are different theories about migraines, but a common theory describes a phenomenon called cortical spreading depression.
Note: This information is based on a well-researched theory. However, this theory is currently the best available. As new research is published, this information will be updated and changed, if necessary.
It is described that migraine headaches occur following a particular trigger. These triggers may include coming in contact with an allergen, certain ingredients in food, physical or psychological stress, weather patterns, lack of sleep, etc.
In the model of cortical spreading depression (CSD), a wave of depolarization (or activation of a nerve) spreads across the cortex of the brain following the trigger of a migraine headache.
Note: the cortex of the brain is where all conscious activity occurs, including conscious thought, speech, motor control, and sensory input.
Following this wave of depolarization, brain activity becomes mildly depressed. In other words, the nerves become inhibited and electrical activity is decreased.
CSD involves a complex series of changes that lead to the release of molecules (namely, potassium, nitric oxide, and adenosine) that irritate the trigeminal nerve endings surrounding the blood vessels in in the meninges—the protective membrane that surrounds the brain and spinal cord.
Migraine headaches tend to have three phases. The first phase is called the prodrome phase, the second is the migraine or headache phase, and the third is called status migranosis.
If you experience migraine headaches, you will likely experience different symptoms in each phase, and you may experience any combination of this long list of symptoms.
Generally, patients describe feeling “uneasy” and can sense a headache is on its way. In this phase, 25% of patients will experience aura—a visual disturbance that may include blurred vision, tunnel vision, or sharp blind spots.
Often times, the aura begins in peripheral vision and expands across the full field of vision. These visual disturbances will likely begin to fade and disappear after approximately 30 minutes
This phase is often characterized by a debilitating, pounding headache—often on only one side of the head. Along with this headache, common symptoms may include:
The symptoms described in the migraine headache phase may persist for 24-48 hours after the onset. Generally, the symptoms will slowly improve until resolution.
Patients often describe this phase as “the hangover,” comparing their symptoms to the way they feel after drinking alcohol.
In our experience, patients have had decreased migraine frequency and decreased symptom severity with upper cervical spinal adjustments and manual therapy of the muscles of the head, neck, and upper back.
For migraine patients, we work with them to identify their triggers. We work together to create a log of their headaches that includes:
Through this log, we work to identify and avoid potential triggers.
Additionally, we suggest supplementing caffeine (200-400mg for adults) daily to help prevent migraine headaches. Steady doses of caffeine, daily, may reduce the frequency and severity of migraine headaches with little-to-no associated risk.
For a migraine that has been triggered (the prodrome phase has begun), you may take an additional 200-400mg of caffeine. Research has indicated caffeine is an effective supplement to help reduce the duration and severity of migraine symptoms. The caffeine tends to be more effective the sooner it is taken following the onset of the prodrome phase.
Note: since migraine headaches are neurological, using non-steroidal anti-inflammatory drugs such as Advil, Aleve, or Tylenol are not effective treatment options.
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This information is for educational purposes only and is not intended to replace the advice of your doctor. Esports Healthcare disclaims any liability for the decisions you make based on this information.
The information contained on this website does not establish, nor does it imply, doctor-patient relationship. Esports Healthcare does not offer this information for diagnostic purposes. A diagnosis must not be assumed based on the information provided.
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